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Problem was, I'd never really achieved that super lean, athletic look with visible abs.
I thought, "Man, I work hard in the gym, and my diet's pretty good too. So why do I still feel uncomfortable taking my shirt off at the pool? I knew that my eating habits were okay, but they weren't perfect. I still struggled to choose the right foods and not overeat. Despite all I'd accomplished, all the weight I'd lost, I still had some residual "fat-guy cravings" for foods that kept me from getting as lean as I wanted.
Sometimes I'd be "good" all week, then blow it all on the weekend with a cheat meal that turned into a hour buffet binge. Again, as a fitness professional it was embarrassing.
Why couldn't I control myself? How come the willpower I expressed in the gym didn't always express itself when it came to my eating habits?
I trained hard too, but that last layer of fat hid the hard-earned muscle. I decided right then to make one last effort to uncover my abs. Since I was already working out several times a week, I knew I had to do something extreme with my diet to shed that stubborn layer of fat.
Being an "insider" in the fitness biz, I had access to a lot of nutrition experts, research scientists, athletes, and bodybuilders: men and women who knew a lot about getting lean very fast without losing muscle. I picked their brains and eventually came up with a strict and honestly quite extreme diet plan, based loosely on Protein-Sparing Modified Fasts, which are dietary regimens very overweight patients at hospitals are put on before they get certain surgical procedures.
I wrote an article about my idea and published it online with a "don't try this at home" warning.
It was an experiment after all. I had no idea how — and if — it would even work.
The Shocking Results In four weeks I lost sixteen pounds of fat and four inches off my waist. My body-fat percentage hit the single digit mark for the first time in my life.
I could see all of my abs. I also noticed a peculiar side effect, too. During the diet I'd somehow lost all my cravings for fattening foods, like sweets and fried stuff. I'd even developed new tastes for healthier foods I'd never liked before, like green vegetables. In the control diet rats, the DBP increased by Figure 1. The bars show the mean; the vertical lines represent the standard error of the mean.
Figure 2. The heart rate increased by Figure 3. These responses lasted longer in the fructose-rich diet rats than in the control diet rats 70  min compared to The fructose-rich diet increased plasma glucose concentrations of the corresponding rats by This increase is much greater than that achieved when diets are fed for shorter periods 9.
Clearly, this longer administration period provides a greater, more prolonged stimulus to the beta cells of the pancreas. The increase in plasma insulin concentrations was insufficient to normalize blood sugar levels due to the simultaneous development of insulin resistance, similar to that seen in diabetes type 2.
The underlying mechanism is a reduction in the synthesis and storage of triglycerides in the adipose tissue provoked by a reduction in the activity of lipoprotein lipase in this tissue, and a reduction in glycogen production at the liver.
In agreement with this finding, obese patients with predominantly visceral fat deposits are reported to show greater sympathetic activity.
The fructose-rich diet induced insulin resistance through the repetitive stimulation of the pancreatic beta cells, a consequence of which is an increase in the plasma insulin concentration. The increased interaction between insulin and its receptors at the target tissues such as the muscles and adipose tissue provokes the autophosphorylation of tyrosine residues in the receptor's internal domains34 as well as reductions in the expression and distribution of cellular glucose transporters.
The increase in sympathetic discharge induced by NaCl is probably 1 of the factors that maintains blood pressure high in the fructose-rich diets rats. One possibility is a sensitizing effect provoked by high plasma insulin levels passing through the AV3V region, where there is no blood-brain barrier.
The AV3V region includes the subfornical organ and the organum vasculosum of the lamina terminalis, which show increased permeability to high molecular weight substances due to the absence of the above barrier.
Long-term fructose rich-diet led to a metabolic state involving insulin resistance and high blood pressure. This diet induces an exaggerated hypertensive response when the AV3V region is stimulated with hypertonic NaCl 1.
Correspondence: Dr. Rosa Facultad de Medicina. Universidad Central de Venezuela.